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Considering the physiological role of selenoproteins in antioxidation and the detrimental effects of oxidative stress in chondrocytes, aberrant selenium metabolism is likely to disrupt cartilage homeostasis and cause arthropathic diseases via dysregulation of redox homeostasis (Fig
A90376 Rabbit polyclonal antibody to GLP-1R for WB and IHC
35 , 7S14S (2003)
BMJ 343 , d4163, (2011)
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Mitochondria as multifaceted regulators of cell death
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